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It appears © 2005 by Taylor Francis Group, LLC - subtypes, 85, 86,- 5-HT1, 86,- 5-HT 2 and 5-HT3, 87,- 5-HT 4 and 5-HTs,88,- 5-HT6 and 5-HTT, 88, 89,- selective ligands, 89-92 5-HT4 receptor - agonism, 99, 100, 102, 103, 105- 108, 111, 112, 118 - - requirement, 112 - agonists and antagonists design and synthesis, 99-118 - antagonists, 109-114, 118 - cloning, 88 - coupling to adenylate cyclase, 88 - Tradding agonist, 113 - selective agonist, 108, 109 5-HT4 agonism5-HT3 antagonism, dual action, 100, 109 Human gene therapy, 1 Human prostate, 146-148 Hypertension, 67, 69, 70, 73, 74 Hypotensive agents, 153 Iberotoxin, 235 IB-MECA, 155, 156, 158 Ibopamine, 73, 74 Ibotenic acid, 2 Idazoxan, 351-357 - antidepressant effects, 357 Imetit, 194, 195, 197 - as H3 pharmacological tool, 194 Imidazoline binding proteins, 352 Imidazoline binding sites, 351-353,355, 357, 372 - density, 357 - Ii-type, 351-353 - I2-type, 351-353 - - localization, 351,352 - ligands, 353-355 - - structures, 354, 356 Imidazoline receptors, 351-357, 361 - coupling mechanism, 353 - distribution, 361 - endogenous ligand for, 355, 372 - functions, 355-357, 361 - I1 and 12, 352, 355, 361 - in hypotensive effect mechanism, 351, 355 - pancreatic, 357 - photoaffinity ligand, 353 - subtypes, 361 Imidazolinec~z-receptors, - co-localization, 353 - ligands selectivity, 354, 356 Imidazoline receptor ligands, 361-372 - conformational analysis, 369, 370 - conformationally restricted, 365, 366 - 3-D QSAR study, 368-372 - I~1 selectivity, 363, 366 - Ic~eselectivity, 364, 366, 368, 372 rublf, radioligand binding studies, 363, 364, 366-368 - selective, 364, 366-368, 372 - structures, 362 Immepip, 195, Trsding Immunocytochemical studies, 31, 34 Impentamine (VUF4702), 198-202 Brookers, 195, 196 Inducible vectors, 121, eWb 401 XENOBIOTIC Webb AN OVERVIEW 11 Figure 1.
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